Download Ciba Foundation Symposium - Homeostatic Regulators by G.E.W. Wolstenholme PDF

By G.E.W. Wolstenholme

Chapter 1 Chairman's advent (pages 1–4): Professor F. Bergel
Chapter 2 telephone inhabitants Kinetics on the subject of Homeostasis (pages 5–15): L. F. Lamerton
Chapter three Chalones of the outside (pages 29–56): Olav Hilmar Iversen
Chapter four Nerve development and Epithelial progress elements (pages 57–74): C. A. Vernon, Barbara E. C. Banks, D. V. Banthorpe, A. R. Berry, H. ff. S. Davies, D. Margaret Lamont, F. L. Pearce and Katharine A. Redding
Chapter five “Wolff elements” from Chick Embryo Mesonephros and Liver or Yeast (pages 75–84): J. Mason, Em. Wolff and Et. Wolff
Chapter 6 Thromboplastic fabrics from Human Tumours and Chorion (pages 85–106): R. A. Q. O'Meara
Chapter 7 development of Gene Transcription through the Induction of Bacteriophage Lambda improvement: a potential version for the regulate of Gene Expression in a Differentiating method (pages 107–127): F. Gros, P. Kourilsky and L. Marcaud
Chapter eight The Histones, their Interactions with DNA, and a few facets of Gene keep an eye on (pages 128–143): E. W. Johns
Chapter nine Enzymes and Isoenzymes (pages 144–164): Cecil L. Leese
Chapter 10 Interferons as attainable Regulators (pages 165–170): N. B. Finter
Chapter eleven Interferons as attainable Regulators—Biochemical points (pages 171–179): D. C. Burke
Chapter 12 Lysosomes and Homeostatic rules (pages 180–196): Pierre J. Jacques
Chapter thirteen Regulatory Mechanisms in Antibody Synthesis (pages 197–229): Gouran Moller
Chapter 14 The constitution of Mammalian mobile Surfaces (pages 230–240): J. A. Forrester
Chapter 15 The mobile Membrane and phone keep watch over (pages 241–263): L. Wolpert and D. Gingell
Chapter sixteen Regulating structures in telephone tradition (pages 264–275): M. G. P. Stoker
Chapter 17 Metabolic Cooperation among Cells (pages 276–290): J. H. Subak?Sharpe
Chapter 18 trend Formation and Homeostasis (pages 291–317): Tom Elsdale
Chapter 19 Chairman's ultimate comments (pages 318–319): Professor F. Bergel

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It is unnecessary to postulate a so-called wound hormone or other stimulating factors, even if we cannot exclude the possibility that such factors also exist. The local character of the regenerative reaction could theoretically be caused also by a nervous mechanism of the axon-reflex type, but then the borderlines of the reaction would have been irregular like “the red flare” in Lewis’s famous “triple reaction’’ (Lewis, 1g27), and have extended much more than I mm outside the damaged area. HISTORICAL BACKGROUND The theoretical background for this way of thinking was specifically formulated by Weiss and Kavanau (1957) when they published a general 35 CHALONBS OF THE SKIN theoretical growth model in 1957.

In the following the practical designation “chalone” will therefore be used for extracts or fractions of extracts of homogenized fresh skin (or other tissues) produced with the methods described. One has to work with the available tests, and from these it is difficult to know whether a certain factor should be called a chalone. The assay systems are also described below. THE PRODUCTION OF CRUDE EPIDERMAL CHALONE The most natural next step was to homogenize epidermis, make extracts, and test the effect of these extracts on the mitotic activity of squamous cell epithelium and of other cell types.

The normal cells retain their normal sensitivity to the growth-controlling signal whereas the cancer cells are less sensitive. Cancer chemotherapy with chalones will theoretically stop the proliferation of the normal cells of that series before it stops the proliferation of the malignant cells. The only possibility for a good effect would be if the mechanism of action of chalones is much more complicated than just inhibition of proliferation, or if it were possible to treat the tumour cells only, a technique that would not be easy to use.

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