Download Atherosclerosis and Oxidant Stress: A New Perspective by James S. Forrester MD (auth.), Professor Jordan L. Holtzman PDF

By James S. Forrester MD (auth.), Professor Jordan L. Holtzman M.D., Ph.D (eds.)

Vascular damage is initiated via oxidant damage to the vessel wall. This quantity is equipped round the position of oxidant harm during this sickness strategy and gives a set of the newest stories, representing quite a few disciplines, which may function the root for extra advancements in our knowing and keep an eye on of cardiovascular and cerebral vascular disease.

Jordan L. Holtzman, M.D., Ph.D. is Professor, Departments of Pharmacology and drugs and department of Environmental healthiness Sciences, collage of Minnesota, Minneapolis, Minnesota.

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Extra resources for Atherosclerosis and Oxidant Stress: A New Perspective

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05). 98). The WHS was the largest trial to date to evaluate clinical outcomes over an extended time period, and the results of this important study did not support the use of vitamin E supplementation for the prevention of CVD among healthy women. 3 Interventional Trials of Antioxidants 31 In summary, trials of vitamin E supplementation have provided inconsistent results in the primary prevention of CVD, with a recent large study (WHS) that failed to show convincing CVD benefit for long-term use of vitamin E.

22 After adjustment for cardiac risk factors, the relative risk (RR) of CHD for those in the highest vs. 01). Further analysis revealed that the protective association was strongest for vitamin E consumed in supplements. 84) for CHD compared with men who did not take vitamin E supplements. 11). 23 In contrast to the NHS and HPFS findings, vitamin E intake from food but not from supplements was strongly associated with a lower risk of CHD mortality. 004). Controlling for other dietary factors associated with vitamin E intake, such as intake of linoleic acid, folate, and fiber did not affect the results.

Hence, alterations in dietary patterns can give important insight into the benefit or harm of nutrients when linked to subsequent changes in markers of oxidative stress. In the DASH trial, consumption of the DASH diet reduced breath ethane exhalation (an in vivo marker of oxidized n-3 polyunsaturated acids)17 and reduced urinary isoprostanes (an in vivo degradation product of arachidonic acid). These findings provide indirect evidence for reduced oxLDL in vivo. 15 Consumption of the DASH diet resulted in increasing serum antioxidants and the ORAC of serum (Fig.

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