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By Andrew D Shaw; et al

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Extra resources for Acute care of the cancer patient

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The pathogenesis of cancer metastasis. Nature 1980; 283:139–146. 6. Heppner G. Tumor heterogeneity. Cancer Res 1984; 44: 2259–2265. 7. Nicolson GL. Generation of phenotypic diversity and progression in metastatic tumors. Cancer Metastasis Rev 1984; 3:25–42. 8. Killion JJ, Kollmorgen GM. Isolation of immunogenic tumor cells by cell-affinity chromatography. Nature 1976; 259:674–676. 9. Kerbel RS. Implications of immunological heterogeneity in tumors. Nature 1979; 280:358–360. 10. Killion JJ. Immunotherapy with tumor subpopulations.

Protein kinases as targets for anticancer agents: from inhibitors to useful drugs. Pharmacol Ther 2002; 93:79–98. 83. Ranson M, Thatcher N. Commentary on ZD1839 (Iressa) in non-small cell lung cancer. Lung Cancer 2003; 40:77–78. 84. Bruns CJ, Harbison MT, Davis DW, Portera CA, Tsan R, McConkey DJ, Evans DB, Abbruzzese JL, Hicklin DJ, Radinsky R. Epidermal growth factor receptor blockade with C225 plus gemcitabine results in regression of human pancreatic carcinoma growing orthotopically in nude mice by antiangiogenic mechanisms.

84) demonstrated regression of human pancreatic carcinoma growing orthotopically in nude mice by therapy with IM-C225 antibody plus gemcitabine. Although both gemcitabine and IM-C225 had activity in this model and are used as single agents, superior results, as measured by tumor burden and incidence of liver metastasis, were achieved by combination therapy. Of particular interest was the finding that apoptosis of tumor-associated endothelial cells was induced by treatment with IM-C225 (due to a coordinate downregulation of VEGF and IL-8 production of the pancreatic carcinoma cells by binding of the IM-C225 antibody).

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